This is unpublished

Hypothalamic gliosis and cardiometabolic disease risk

December 15, 2023
Ellen Schur, MD, MS


Upon completion of this program, attendees should be able to:

  • Gain familiarity with the actions of glial cell populations in the central nervous system and preclinical studies demonstrating that hypothalamic inflammation and gliosis promote diet-induced obesity
  • Comprehend accumulated translational evidence that hypothalamic inflammation and gliosis are present in humans with obesity and type 2 diabetes
  • Appreciate potential roles for hypothalamic inflammation and gliosis in CVD pathogenesis in humans


The University of Washington School of Medicine is accredited by the Accreditation Council for Continuing Medical Education to provide continuing medical education for physicians.

The University of Washington School of Medicine designates this live activity for a maximum of 36.0 AMA PRA Category 1 Credits.

Physicians should claim only the credit commensurate with the extent of their participation in the activity. (Each session is 1.0 credit)

To request disability accommodation, contact the Office of the Disability Services Office at least ten days in advance of the event at 206-543-6450 (voice), 206-543-6452 (TTY), 206-685-7264 (FAX),